CalcMyPeptide
Cognitive & NootropicAlso known as: N-hexanoic-Tyr-Ile-(6) aminohexanoic amide

Dihexa

Dihexa is a synthetically engineered Angiotensin IV analog originally developed to combat aggressive neurodegenerative diseases like Alzheimer's and Parkinson's. Acclaimed for having a synaptic formation potency literally 10 million times stronger than Brain-Derived Neurotrophic Factor (BDNF), Dihexa is the ultimate heavy-weight nootropic. In elite neuro-optimization contexts, it is utilized to profoundly accelerate learning, permanently repair traumatized neural pathways, and dramatically enhance raw cognitive neuroplasticity.

Reviewed by CalcMyPeptide Editorial Team
Last updated: April 2026Evidence: Low1 peer-reviewed citation

Quick Stats

Half-LifeUnknown (central effects persist >48h)
Dose Range10-40 mg/day (oral/intranasal)
Frequency1× daily
Vial SizesN/A (oral)
BioavailabilityOral and intranasal — crosses blood-brain barrier
Year Developed2011

Scientific Data

Molecular Formula
C21H33N3O4
Molecular Weight
407.51 g/mol
CAS Number
Developer
Joe W. Harding & John W. Wright, Washington State University

Mechanism of Action

Dihexa (N-hexanoic-Tyr-Ile-(6)-aminohexanoic amide) is a synthetic peptide analog of angiotensin IV that acts on hepatocyte growth factor (HGF) receptors. It is reported to be 10^7 (ten million) times more potent than BDNF (Brain-Derived Neurotrophic Factor) at promoting spinogenesis — the formation of new dendritic spines that enable synaptic connections.

Dihexa crosses the blood-brain barrier and promotes synaptogenesis, which may enhance learning, memory, and cognitive function. It was developed at Washington State University as a potential therapeutic for neurodegenerative diseases including Alzheimer's.

Source: PMID: 23123685

Background & History

Dihexa is a potent cognitive enhancer derived from angiotensin IV, developed by Joseph Harding and John Wright at Washington State University. Published in Science (2012), it was shown to induce synaptogenesis — the formation of new synapses — with potency reportedly 7 orders of magnitude greater than BDNF in some assays. It acts via HGF/MET receptor signaling.

Research Use Cases

  • Cognitive decline in Alzheimer's disease and dementia (animal models)
  • Post-stroke cognitive recovery
  • Traumatic brain injury recovery
  • Nootropic stack component for memory and learning enhancement

Dosing Protocol

Typical Dose10-40 mg/day (oral/intranasal)
Frequency1× daily
Half-Life~2-4 hours (estimated)

Dosing Protocols

Standard Protocol

Dose
10 - 40 mg
Frequency
Daily (oral or intranasal)
Note: Extremely potent. Effects persist >48h. Not used by SC injection.

Administration

Route
Oral or intranasal
Timing
Morning. No fasting requirement.
Fasting Required?
No — food timing not critical

Expected Timeline

Days to weeks
Anecdotal cognitive clarity improvements, enhanced learning and memory formation.
Week 4+
Synaptogenesis effects are cumulative — most users report compounding cognitive benefit.

Who Is It For?

Cognitive Enhancement / Nootropic

Low

10 million times more potent than BDNF at spinogenesis in vitro. Very limited human data.

Neurodegenerative Disease Research

Low

Developed for Alzheimer's research. Powerful but data is almost entirely preclinical.

Safety & Considerations

Research peptide with very limited human data. Extremely potent — doses measured in mg/day. Oral or intranasal only. Consult a healthcare provider before use.

Regulatory & Legal Status

FDA Status (US)
Research Only
WADA Status (2026)
Not Listed

Not currently on the WADA 2026 Prohibited List. Policies may change — verify before competition.

Classification

Research Chemical

US Compounding: Not eligible / not available

⚠️ This information is for educational purposes only and may not reflect the most current regulatory updates. Always verify with official FDA, WADA, and jurisdiction-specific sources before use.

Interactions & Contraindications

Long-acting — effects may persist for weeks after single dosing. Start at very low doses. Limited human safety data available. Avoid with active brain tumors.

Synergies & Common Stacks

Semax upregulates BDNF; Dihexa promotes synaptogenesis via HGF/MET. Together they support neurotrophic support from multiple angles in cognitive recovery protocols.

Cerebrolysin contains BDNF, NGF, and other neurotrophins; Dihexa promotes the synaptic infrastructure to utilize them. Potentially synergistic for neurological recovery.

Dosing Quick Reference

Dihexa— Dosing Guide
Dose Range
10-40 mg/day (oral/intranasal)
Half-Life
~2-4 hours (estimated)
Frequency
1× daily
Route
Oral
Cognitive & Nootropiccalcmypeptide.com

Frequently Asked Questions

Is Dihexa injectable?
No — typically oral or intranasal at 10-40 mg/day. No reconstitution needed. Not used as a subcutaneous injection.
How potent is Dihexa vs BDNF?
Reported to be 10 million times more potent at spinogenesis in vitro. This is receptor-specific potency, not a general nootropic efficacy comparison.

References

  1. Bhargava A et al. "Facilitating effects of a novel angiotensin IV analogue on recovery from traumatic brain injury".” Journal of Cognitive Neuroscience (2011). PMID: 21544069

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