Mitochondrial

SS-31

Elamipretide · Bendavia · MTP-131 · Forzinity

Elamipretide. Forzinity is FDA accelerated-approved (Sep 2025) for Barth syndrome ≥30 kg - that product is not a 5 mg research cake. Cardiolipin-targeted tetrapeptide. Mixed cardiomyopathy trial history. Not a general anti-aging serum.

Reviewed by CalcMyPeptide Editorial Team
Last updated: August 2026Evidence: Moderate3 peer-reviewed citations
Typical research dose
20-40 mg/day (clinical trials)
Frequency
1× daily
Half-life
~4-5 hours
Common vials
5 mg
CAS
736992-21-5
Molecular weight
736.83 g/mol

Use-case scores

0–10 against the evidence we have, not a gym ranking. Hidden domains do not apply here.

  • Longevity / aging4/10

    Forzinity is FDA accelerated-approved (Sep 2025) for Barth syndrome ≥30 kg. That is a rare-disease label, not an anti-aging NDA. Gray-market SS-31 is not Forzinity. Haircut to 4.

  • Injury & tissue3.5/10

    Ischemia and cardiomyopathy trial history is mixed. BENEFIT-class heart-failure programs are not a gym recovery protocol.

Compound card

Sequence
D-Arg-Dmt-Lys-Phe-NH2
Class
Mitochondria-targeted tetrapeptide (elamipretide; Forzinity is the labeled Barth product)
Formula
C32H52N10O8
Status
FDA-approved
Dose it
5 mg + 2 mL BAC water → 2.5 mg/mL · 20000 mcg ≈ 800 units (U-100)
Open in calculator

How it works

SS-31 (Elamipretide, Bendavia) is a mitochondria-targeting tetrapeptide (D-Arg-Dmt-Lys-Phe-NH2) that selectively binds to cardiolipin on the inner mitochondrial membrane. Cardiolipin is essential for electron transport chain function - when it becomes oxidized with age, mitochondrial efficiency declines. SS-31 prevents cardiolipin peroxidation, restoring mitochondrial bioenergetics.

SS-31 is unique among peptides in that it concentrates 1000-5000× in mitochondria versus the extracellular space. It is in clinical trials for Barth syndrome, age-related macular degeneration, and primary mitochondrial myopathy.

Source: PMID: 26857943

Background & History

SS-31 (Elamipretide, MTP-131) is a tetrapeptide targeting cardiolipin - a phospholipid exclusively located in the inner mitochondrial membrane critical for electron transport chain function. Developed by Hazel Szeto at Weill Cornell Medical College in the 2000s, SS-31 penetrates the inner mitochondrial membrane (unique for a water-soluble peptide) and protects cardiolipin from oxidative damage. Clinical trials have evaluated it in heart failure (BENEFIT-HF), kidney disease, and Barth syndrome. It is the most clinically advanced mitochondria-targeted therapeutic peptide.

Research Use Cases

  • ✓Identity: Forzinity Barth label vs a research cake
  • ✓Cardiolipin targeting, not a general anti-aging serum
  • ✓Why mixed cardiomyopathy programs matter

Dosing

PhaseDoseFrequencyNotes
Research Protocol0.1 - 0.25 mg/kgDaily or 5x weekly (SC)Concentrates 1000-5000x in mitochondria. Clinical trials ongoing for Barth syndrome and AMD.

Administration

Route / form
Subcutaneous injection
Timing
Morning. Fasting not required.
Empty stomach?
No - Food timing is not critical.

Timeline

Week 1-4
Improved cellular energy. Reduced oxidative stress markers.
Month 1-3
Functional improvements in mitochondrial disease models. Exercise capacity improvements.

Who it is for

Mitochondrial Dysfunction

High

Directly targets inner mitochondrial membrane cardiolipin. In clinical trials for primary mitochondrial diseases.

Longevity / Anti-Aging

Moderate

Restores age-associated mitochondrial bioenergetic decline.

Reconstitution

VialWaterConcentrationExample dose
5 mg lyophilized2 mL BAC2.5 mg/mL5 mg / 2 mL = 2.5 mg/mL. Trial doses were tens of mg/day. Community 5 mg cakes are not Forzinity.

Change vial size or water volume? Open the reconstitution calculator.

Safety & Considerations

Investigational drug in active clinical trials. Not FDA-approved. Generally well-tolerated. Injection site reactions are the most common adverse event.

Regulatory & Legal Status

FDA Status (US)
Approved

Accelerated approval Sep 2025 as Forzinity™ (elamipretide) for Barth syndrome ≥30 kg. Research vials and stacks are not that product

WADA Status (2026)
Not Listed

Not currently on the WADA 2026 Prohibited List. Policies may change - verify before competition.

Classification

Prescription Drug

US Compounding: Not eligible / not available

⚠️ This information is for educational purposes only and may not reflect the most current regulatory updates. Always verify with official FDA, WADA, and jurisdiction-specific sources before use.

Limits of current evidence

  • Do not paste Forzinity Barth dosing onto a research vial.
  • Failed or mixed cardiomyopathy programs are part of the file. Cherry-picking open-label Barth data is dishonest.
  • This site will not treat heart failure, kidney injury, or aging from this page.

Human evidence

Moderate · Barth-syndrome label story; gray cake ≠ Forzinity

  • Elamipretide reached rare-disease labeling for Barth syndrome (Forzinity) — that is not an anti-aging NDA.
  • A 5 mg research cake is not Forzinity trial or labeled supply.
  • Cardiomyopathy and related programs are mixed; do not cherry-pick open-label Barth data as a gym protocol.

Verdict

SS-31 is elamipretide. Forzinity is the Barth-syndrome product. A 5 mg research cake is not that label and not a general mitochondrial youth serum. Cardiolipin targeting is the mechanism, not a lifespan claim.

Interactions & Contraindications

Forzinity is the Barth product. Gray-market SS-31 is not that label. SubQ research cakes are not an oral vitamin. Do not treat heart failure from this page.

Synergies & Common Stacks

Cardiolipin tetrapeptide vs AMPK mtDNA ORF. Complementary mitochondrial marketing is not a combination trial.

NAD+ supports NADH/NAD+ cycling; SS-31 protects the complexes that use NADH. Additive protection of the electron transport chain.

Frequently Asked Questions

What is SS-31 used for?▼
SS-31 (Elamipretide) targets the inner mitochondrial membrane to restore bioenergetics. In clinical trials for Barth syndrome, primary mitochondrial myopathy, and age-related macular degeneration.
How does SS-31 differ from MOTS-c?▼
SS-31 concentrates directly in mitochondria (1000-5000x) by binding cardiolipin. MOTS-c acts via AMPK signaling. Fundamentally different mechanisms - sometimes combined.

References

  1. Szeto HH “"Elamipretide (SS-31) improves mitochondrial dysfunction and muscle function".” Journal of Bioenergetics and Biomembranes (2013). PMID: 24337793
  2. Szeto HH. “SS-31 peptide enables mitochondrial targeting drug delivery.” AAPS J (2006). PMID: 17233531
  3. Siegel MP, et al. “Mitochondria-targeted peptide SS-31 reverses mitochondrial dysfunction and reduces oxidative stress.” Aging Cell (2013). PMID: 23834033

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